Cardiac Risk Markers is available for online booking with home sample collection.
Cardiac Risk Markers Info & Procedure
Group NameWELLNESS
FastingOvernight Fasting required
Sample TypeBlood (Serum)
Sample CollectionHome sample collection
Parameters5 included
LaboratoryThyrocare
Tests included
5 parameters listed
Explore the tests in Cardiac Risk Markers by category.
Cardiac Risk Markers5
APO B / APO A1 RATIO (APO B/A1)
APOLIPOPROTEIN - A1 (APO-A1)
APOLIPOPROTEIN - B (APO-B)
HIGH SENSITIVITY C-REACTIVE PROTEIN (HS-CRP)
Lipoprotein (a) [Lp(a)]
Cardiac Risk Markers Sample Report Format & Ranges
Relevant details from a Thyrocare sample report are shown below. A standalone PDF preview for Cardiac Risk Markers is not available here.
Reference intervals may vary by method and patient factors. Use the interval printed on your own final report.
Conducted at
Thyrocare Labs
Lab Accreditation
CAP, NABL, UKAS, ANAB
Cardiac Risk Markers Thyrocare Sample-Report Range Table
Parameter
Unit
Reference Range
FASTING BLOOD SUGAR(GLUCOSE)
mg/dL
70-100
HbA1c
%
< 5.7
AVERAGE BLOOD GLUCOSE (ABG)
mg/dL
90-120
Cardiac Risk Markers Report Sections Covered
Report Section
Parameters in Sample Report
Vitamin
2
Iron Deficiency
4
Lipid
9
Liver
12
Electrolytes
3
Renal
6
Thyroid
1
Cardiac Risk Markers
1
Diabetes
3
Complete Hemogram
28
Profile visual guide
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Cardiac Risk Markers Reference Guide
Cardiac Risk Markers combines five serum measurements for cardiovascular risk refinement.
ApoB estimates atherogenic particle burden, while ApoA1 adds HDL-associated context.
Lp(a) contributes a largely inherited risk signal.
Hs-CRP adds non-specific inflammation context during clinically stable periods.
The profile supplements standard lipids, blood pressure, diabetes and smoking assessment.
Why This Test Is Ordered
Reason
Description
Estimate atherogenic particle burden with ApoB
ApoB is present on LDL and other particles that can enter artery walls.
Each major atherogenic particle carries one ApoB molecule.
ApoB therefore estimates particle number rather than cholesterol mass.
Particle number and LDL cholesterol may not always align closely.
Discordance can matter with diabetes, metabolic syndrome or high triglycerides.
Higher ApoB can strengthen concern about plaque-forming particle burden.
ApoB cannot show whether an artery is narrowed or blocked.
Standard LDL, non-HDL cholesterol and triglycerides remain important.
Treatment decisions also depend on age, history and overall risk.
Compare ApoB with the HDL-associated ApoA1
ApoA1 is the main protein component of high-density lipoprotein particles.
It contributes to cholesterol transport within the broader lipid system.
The ratio compares ApoB with the HDL-associated ApoA1 measurement.
Either component can change the calculated ApoB-to-ApoA1 ratio.
A less favourable balance may add information about cardiovascular risk.
The ratio is not an independent substance or a diagnosis.
Use the laboratory method and current report units for interpretation.
Standard cholesterol results remain necessary for prevention decisions.
Identify a largely inherited Lp(a) risk signal
Lp(a) is a cholesterol-carrying particle containing ApoB and apolipoprotein(a).
The Lp(a) level is determined mainly by inherited genetic variation.
Levels often remain fairly stable through adult life.
Higher Lp(a) can add risk for heart disease and stroke.
Risk may remain relevant even when LDL cholesterol is well controlled.
Current guidance supports at least one adult Lp(a) measurement.
Family history of premature disease makes inherited risk especially relevant.
Reports may use mg/dL or nmol/L, which are not interchangeable.
Read the result in its stated unit without fixed-factor conversion.
Add inflammation context with hs-CRP
Hs-CRP measures small concentrations of C-reactive protein in blood.
The liver produces CRP in response to inflammation within the body.
Hs-CRP can act as a risk-enhancing marker during cardiovascular prevention.
The marker is not specific to inflammation inside artery walls.
Colds, fever, dental infection, injury or surgery can raise it.
Inflammatory conditions may also produce a temporarily higher result.
An unexpected elevation may need reassessment after recovery.
Hs-CRP cannot locate inflammation or diagnose coronary disease.
Use the panel to refine, not replace, standard risk assessment
Cardiovascular risk includes age, blood pressure, diabetes and smoking history.
Standard cholesterol, kidney health and family history also remain important.
This package adds selected risk markers without calculating risk percentage.
Total cholesterol, LDL, HDL and triglycerides are not included.
Glucose and HbA1c results are also outside this package.
The profile cannot show plaque, coronary calcium or blood flow.
A clinician may add lipids, diabetes testing or blood-pressure review.
Five normal markers cannot cancel hypertension, smoking or diabetes risk.
One abnormal marker cannot predict that a heart attack will occur.
Symptoms And Who Should Test
Situation
Description
People with premature cardiovascular disease in the family
An early heart attack or stroke in a close relative can prompt review.
Record the relative’s age, diagnosis and known cholesterol disorder.
Lp(a) is relevant because its concentration is largely inherited.
ApoB may reveal particle burden not fully described by LDL cholesterol.
This profile does not test genes for familial hypercholesterolaemia.
It also does not replace a standard LDL cholesterol measurement.
Higher Lp(a) may justify discussing testing with close relatives.
Modifiable risk factors still need attention when inherited risk is present.
People with diabetes, high triglycerides or metabolic syndrome
Insulin resistance can alter the cholesterol carried inside atherogenic particles.
ApoB may add information when LDL cholesterol appears acceptable.
Type 2 diabetes and high triglycerides can create particle discordance.
Blood pressure and glucose control remain central to prevention.
HbA1c, kidney function and standard lipids are not included here.
Hs-CRP may rise with obesity or another inflammatory condition.
Hs-CRP is not a direct measurement of arterial plaque.
Testing should refine a defined prevention or treatment decision.
People with established heart or arterial disease
Previous heart attack or ischaemic stroke already indicates high clinical risk.
Peripheral arterial disease or coronary procedures also establish important history.
ApoB may help assess residual particle burden during lipid-lowering treatment.
Lp(a) can add information about inherited residual risk.
These markers do not replace treatment targets chosen by the cardiologist.
Do not independently change statins or other medicines from this report.
A lower hs-CRP does not mean established plaque has disappeared.
Share medicine adherence and recent lipid reports with the reviewer.
New chest pressure or stroke symptoms require emergency care.
People whose standard risk estimate remains uncertain
Standard assessment starts with age, blood pressure, cholesterol and diabetes.
Smoking and family history add further information to the risk estimate.
ApoB, Lp(a) and hs-CRP can refine selected prevention decisions.
The panel is not automatically necessary at every routine check.
No single result dictates one treatment without clinical discussion.
Expected benefit, possible harms and patient preferences still matter.
Coronary calcium imaging may answer a different question in some adults.
Testing should focus on the uncertainty that remains after standard assessment.
People planning a broader preventive review
High cholesterol and high blood pressure often cause no symptoms.
A person who feels well may still have cardiovascular risk.
This profile can support a clinician-led preventive review.
It should accompany measured blood pressure and a standard lipid profile.
Smoking, activity, diet, weight and diabetes require separate review.
Kidney health and chronic inflammation may also alter risk.
Age, reproductive history and family history add personal context.
The goal is reducing overall heart attack and stroke risk.
Preparation And Interpretation Notes
Topic
Guidance
Fasting and serum collection
Fasting is not required for these five serum markers.
Follow stricter preparation when another booked test requires fasting.
A trained professional collects blood from a vein and prepares serum.
Maintain ordinary hydration unless a clinician advises otherwise.
Do not prolong fasting or change food intake to influence results.
Minor pain or bruising can follow routine blood collection.
Tell the collector about fainting or difficult previous collections.
Mention blood-thinning medicine before the sample is collected.
These instructions do not apply automatically to every cardiac blood test.
Choose a clinically stable time for hs-CRP
Hs-CRP responds to inflammation arising from many different causes.
Report fever, respiratory infection or a current dental infection.
Also mention vaccination, injury, surgery or an autoimmune flare.
A temporary elevation may not reflect the usual cardiovascular-risk signal.
The clinician may defer testing or repeat it after recovery.
Do not delay treatment of infection to obtain a lower result.
A low hs-CRP result should not dismiss active symptoms.
Hs-CRP cannot diagnose infection or an acute heart problem.
Report medicines, supplements and health changes
List lipid-lowering medicines, anti-inflammatory drugs and hormone therapy.
Include all other prescriptions, supplements and recent dose changes.
Do not stop statins or aspirin without prescriber advice.
Treatment can influence ApoB and inflammation-related measurements.
Pregnancy, kidney disease and chronic inflammation affect interpretation context.
Substantial weight change or recent hospitalisation should also be reported.
Bring earlier lipid and biomarker reports when available.
State whether testing is baseline, follow-up or inherited-risk assessment.
Keep Lp(a) units and repeat testing in perspective
Lp(a) may be reported in mg/dL or nmol/L.
The units describe different properties and are not interchangeable.
Do not convert Lp(a) using one fixed universal multiplier.
Use the current laboratory’s unit and decision threshold.
Lp(a) is largely genetic and often needs measurement only once.
A clinician may repeat it after a relevant clinical or treatment change.
ApoB and hs-CRP may be repeated for different reasons.
Keep dated reports to identify method, unit and inflammation differences.
Interpret results through an overall prevention plan
Use the reference information and units on the current report.
Reference ranges and clinical decision thresholds are not always identical.
Review ApoB, ApoA1 and their calculated ratio together.
Then combine Lp(a) and hs-CRP with standard risk factors.
Ask whether results change lifestyle or lipid-lowering priorities.
Do not start aspirin, statins or supplements from this report alone.
Troponin, ECG and cardiac imaging are not included.
This prevention profile cannot diagnose an acute heart attack.
Chest pressure, breathlessness or stroke symptoms need emergency assessment.
Frequently Asked Questions
What does Cardiac Risk Markers include?
It includes ApoA1, ApoB, their ratio, hs-CRP and lipoprotein(a).
Is fasting required for this package?
No; fasting is not required unless another test in the booking needs it.
Is this package the same as a lipid profile?
No; LDL, HDL, total cholesterol and triglycerides are not included.
What does ApoB add to heart-risk assessment?
ApoB estimates the number of atherogenic lipoprotein particles in blood.
Is lipoprotein(a) inherited?
Yes; Lp(a) is largely genetically determined and often remains fairly stable.
Can infection affect hs-CRP?
Yes; infection, injury or inflammation can temporarily raise hs-CRP.
Can this panel diagnose blocked arteries?
No; it refines risk and cannot show plaque or arterial narrowing.
Should I stop statins before testing?
No; do not stop prescribed medicine unless your clinician instructs you.
Can I book home collection?
Home collection is available where the entered address is serviceable.
When should these markers be repeated?
Repeat timing depends on the marker, treatment and clinician’s prevention plan.
Who should review this cardiac-risk report?
A clinician should combine it with lipids, blood pressure and medical history.
Get tested for Cardiac Risk Markers at fully automated diagnostic laboratory Thyrocare which has Centralized Processing Lab (CPL) for esoteric tests and Regional Processing Labs in major cities of India.
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